|
|
||||||||
From the Department of Psychiatry and Behavioral Sciences (Dr. Perman), and the Department of Neurology (Dr. Racy), George Washington University Medical Center, Washington, D. C.
Homolateral ataxia and crural paresis is a recognized vascular syndrome. However, confirmation of the causative lesion rests principally on one earlier case with multiple other infarcts. We studied a patient with the clinical syndrome; computerized tomography revealed a lucency that appeared within 1 week of the infarct. Localization of the lesion to the superior portion of the posterior limb of the internal capsule and thalamus is in accord with the original conclusions of Fisher and Cole.1
Address correspondence and reprint requests to Dr. Racy, Department of Neurology, 2150 Pennsylvania Avenue, N. W., Washington, DC 20037.
Accepted for publication December 10, 1979.
This article has been cited by other articles:
![]() |
N. Nagaratnam, C. Xavier, and R. Fabian Stroke Subtype--Ataxic Hemiparesis Neurorehabil Neural Repair, June 1, 1999; 13(2): 149 - 153. [Abstract] [PDF] |
||||
![]() |
M. J. Gorman, R. Dafer, and S. R. Levine Ataxic Hemiparesis : Critical Appraisal of a Lacunar Syndrome Stroke, December 1, 1998; 29(12): 2549 - 2555. [Abstract] [Full Text] [PDF] |
||||
![]() |
K. Ichikawa, A. Tsutsumishita, and A. Fujioka Capsular Ataxic Hemiparesis: A Case Report Arch Neurol, September 1, 1982; 39(9): 585 - 586. [Abstract] [PDF] |
||||
![]() |
V. J. Iragui and C. B. McCutchen Capsular Ataxic Hemiparesis Arch Neurol, August 1, 1982; 39(8): 528 - 529. [Abstract] [PDF] |
||||
| HOME | HELP | FEEDBACK | SUBSCRIPTIONS | ARCHIVE | SEARCH | TABLE OF CONTENTS |