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NEUROLOGY 1985;35:1146
© 1985 American Academy of Neurology

Encephalomyeloneuritis simulating Guillain-Barré syndrome

H. Robert Brashear, MD, José M. Bonnin, MD and Ivan S. Login, MD

Departments of Neurology and Pathology, Division of Neuropathology, University of Virginia School of Medicine, Charlottesville, VA.

A patient with encephalomyeloneuritis (EMN) had clinical and laboratory features consistent with severe acute inflammatory polyneuropathy (Guillain-Barrk syndrome). CNS involvement was suggested clinically only by transient downbeat nystagmus and by contraction of tensor fascia lata on plantar stimulation. Postmortem examination revealed pathologic changes typical of EMN without systemic neoplasm. Clinical manifestations of the profound central pathology were largely obscured by severe radiculoneuropathy. Pathologic verification of clinically diagnosed inflammatory polyneuropathy is unusual, and CNS disease, therefore, may be more frequent than appreciated, especially in clinical "variants. "

Address correspondence and reprint requests to Dr. Login, Department of Neurology, Box 394, University of Virginia School of Medicine, Charlottesville, VA 22908.

Accepted for publication November 30, 1984.




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