Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Hijdra, A.
Right arrow Articles by Van Crevel, H.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Hijdra, A.
Right arrow Articles by Van Crevel, H.
NEUROLOGY 1986;36:329
© 1986 American Academy of Neurology

Delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage

Clinicoanatomic correlations

A. Hijdra, J. Van Gijn, S. Stefanko, K. J. Van Dongen, M. Vermeulen and H. Van Crevel

Departments of Neurology (Drs. Hijdra, Van Gijn, and Vermeulen), Pathology (Dr. Stefanko), and Radiology (Dr. Van Dongen), University Hospital "Dijkzigt," Rotterdam; the Department of Neurology (Drs. Hijdra and Van Crevel), Academisch Medisch Centrum, Amsterdam; and the Department of Neurology (Dr. Van Gijn), State University, Utrecht, The Netherlands.

Fifty-seven of 176 prospectively studied patients with aneurysmal subarachnoid hemorrhage (SAH) developed delayed cerebral ischemia. Clinical features included hemispheric focal signs (13), decrease in level of consciousness (14), or both (30), and mutism (15). Forty-seven patients showed hypodense lesions on CT in one (19) or multiple vascular territories (22), or diffusely in one or both hemispheres (6). Twenty-eight had symmetric decrease in ventricular size. Of 18 autopsied patients, only 1 had a purely univascular lesion. The clinical, CT, and pathologic features suggest that delayed cerebral ischemia after SAH is a multivascular or diffuse process in most patients.

Address correspondence and reprint requests to Dr. Hijdra, Department of Neurology, Academisch Medisch Centrum, 1105 AZ, Amsterdam, The Netherlands.

Accepted for publication June 27, 1985.




This article has been cited by other articles:


Home page
 BrainHome page
J. P. Dreier, J. Woitzik, M. Fabricius, R. Bhatia, S. Major, C. Drenckhahn, T.-N. Lehmann, A. Sarrafzadeh, L. Willumsen, J. A. Hartings, et al.
Delayed ischaemic neurological deficits after subarachnoid haemorrhage are associated with clusters of spreading depolarizations
Brain, December 1, 2006; 129(12): 3224 - 3237.
[Abstract] [Full Text] [PDF]

Home page
 StrokeHome page
A. A. Rabinstein, S. Weigand, J. L.D. Atkinson, and E. F.M. Wijdicks
Patterns of Cerebral Infarction in Aneurysmal Subarachnoid Hemorrhage
Stroke, May 1, 2005; 36(5): 992 - 997.
[Abstract] [Full Text] [PDF]

Home page
 StrokeHome page
W. M. van den Bergh and on behalf of the MASH Study Group
Magnesium Sulfate in Aneurysmal Subarachnoid Hemorrhage: A Randomized Controlled Trial
Stroke, May 1, 2005; 36(5): 1011 - 1015.
[Abstract] [Full Text] [PDF]

Home page
 BrainHome page
J. van Gijn and G. J. E. Rinkel
Subarachnoid haemorrhage: diagnosis, causes and management
Brain, February 1, 2001; 124(2): 249 - 278.
[Abstract] [Full Text] [PDF]

Home page
 NeurologyHome page
E. H. Brilstra, G. J. E. Rinkel, A. Algra, and J. van Gijn
Rebleeding, secondary ischemia, and timing of operation in patients with subarachnoid hemorrhage
Neurology, December 12, 2000; 55(11): 1656 - 1660.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1986 by AAN Enterprises, Inc.