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NEUROLOGY 1986;36:1192
© 1986 American Academy of Neurology

AIDS

Calcification of the basal ganglia in infants and children

Anita Lesgold Belman, MD, George Lantos, MD, Dikran Horoupian, MD, Brian E. Novick, MD, Monica H. Ultmann, MD, Dennis W. Dickson, MD and Arye Rubinstein, MD

Department of Neurology (Dr. Belman), School of Medicine, State University of New York, Stony Brook, New York; and the Departments of Pathology (Neuropathology) (Drs. Horoupian and Dickson), Radiology (Dr. Lantos), and Pediatrics (Drs. Novick, Ultmann, and Rubinstein), Albert Einstein College of Medicine, Montefiore Hospital and Medical Center, Bronx, NY.

CT or postmortem examination demonstrated calcification of the basal ganglia in eight infants and children with acquired immune deficiency syndrome. Serial CT studies documented progression of both bilateral symmetric calcium densities and cerebral atrophy. Clinical features included progressive encephalopathy with dementia, and pyramidal tract signs. Postmortem examination of four children revealed variable degrees of calcific vasopathy of the basal ganglia, involving predominantly the putamen and globus pallidus.

Address correspondence and reprint requests to Dr. Belman, Department of Neurology, School of Medicine, Health Sciences Center, T12–020, SUNY, Stony Brook, NY 11794.

Supported in part by the New York State AIDS Institute (B.E.N.); United Cerebral Palsy Research and Educational Foundation Clinical Fellowship in Pediatrics (grant no. CF118-84-85). National Institutes of Health, Human Developmental Biology Training Grant (grant no. 5T32HD07053, 1984–85) (M.H.U.); and the National Institutes of Health (grant no. AI20671) (A.R.).

Presented in part at the thirty-seventh annual meeting of the American Academy of Neurology, Dallas, TX, April 1985, and the thirty-eighth annual meeting of the American Academy of Neurology, New Orleans, LA, April 1986.

Accepted for publication December 28, 1985.




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