Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Fross, R. D.
Right arrow Articles by Calne, D. B.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Fross, R. D.
Right arrow Articles by Calne, D. B.
NEUROLOGY 1987;37:1125
© 1987 American Academy of Neurology

Lesions of the putamen

Their relevance to dystonia

R. D. Fross, MD, W. R. W. Martin, MD, FRCPC, D. Li, MD, FRCPC, A. J. Stoessl, MD, FRCPC, M. J. Adam, PhD, T. J. Ruth, PhD, B. D. Pate, PhD, K. Burton, MBBS, FRACP and D. B. Calne, DM, FRCP, FRCPC

Division of Neurology (Drs. Fross, Martin, Stoessl, Burton, and Calne), Department of Medicine, the Department of Radmlogy (Dr. Lil, and the UBC/TRIUMF PET Program (Drs. Adam, Ruth, and Pate), University of British Columbia Faculty of Medicine, Health Sciences Centre Hospital, Vancouver, British Columbia, Canada.

Two patients with acquired dystonia were studied by computed imaging techniques and found to have lesions predominantly involving the putamen. The implications of these findings are discussed, and it is concluded that, for the genesis of dystonia, a relative increase of other inputs to the pallidum may be important, such as those from the caudate and subthalamic nuclei.

Address correspondence and reprint requests to Dr. Calne, Division of Neurology. Department of Medicine, University of British Columbia Faculty of Medicine, Health Sciences Centre Hospital, 2211 Wesbrook Mall, Vancouver, British Columbia, Canada V6T 1W5.

Supported by the Dystonia Medical Research Foundation and the Medical Research Council of Canada.

Received June 5, 1986. Accepted for publication in final form October 13, 1986.




This article has been cited by other articles:


Home page
 J. Neurol. Neurosurg. PsychiatryHome page
C Chuang, S Fahn, and S J Frucht
The natural history and treatment of acquired hemidystonia: report of 33 cases and review of the literature
J. Neurol. Neurosurg. Psychiatry, January 1, 2002; 72(1): 59 - 67.
[Abstract] [Full Text] [PDF]

Home page
 BrainHome page
S. Yazawa, A. Ikeda, R. Kaji, K. Terada, T. Nagamine, K.-i. Toma, T. Kubori, J. Kimura, and H. Shibasaki1
Abnormal cortical processing of voluntary muscle relaxation in patients with focal hand dystonia studied by movement-related potentials
Brain, July 1, 1999; 122(7): 1357 - 1366.
[Abstract] [Full Text] [PDF]

Home page
 J. Neurosci.Home page
J. S. Perlmutter, M. K. Stambuk, J. Markham, K. J. Black, L. McGee-Minnich, J. Jankovic, and S. M. Moerlein
Decreased [18F]Spiperone Binding in Putamen in Idiopathic Focal Dystonia
J. Neurosci., January 15, 1997; 17(2): 843 - 850.
[Abstract] [Full Text] [PDF]

Home page
 Arch NeurolHome page
S. Lehericy, M. Vidailhet, D. Dormont, L. Pierot, J. Chiras, P. Mazetti, C. Marsault, and Y. Agid
Striatopallidal and Thalamic Dystonia: A Magnetic Resonance Imaging Anatomoclinical Study
Arch Neurol, March 1, 1996; 53(3): 241 - 250.
[Abstract] [PDF]

Home page
 Arch NeurolHome page
L. A. Stone and J. Jankovic
The Coexistence of Tics and Dystonia
Arch Neurol, August 1, 1991; 48(8): 862 - 865.
[Abstract] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1987 by AAN Enterprises, Inc.