|
|
||||||||
Divisions of Neurology and Neuropathology, West Roxbury Veteran's Administration Hospital, West Roxbury, MA.
A patient presented with severe sensory loss and ataxia with total arreflexia, and elevation of CSF protein with pleocytosis. At autopsy there was extensive lymphocytic infiltration of nerves and posterior roots, sparing the anterior roots. Teased fiber preparation of nerve showed a demyelinating lesion. There were no abnormalities in the CNS. The condition appears to be an acute sensory polyneuritis bearing a close relationship to acute Guillain-Barré syndrome.
Address correspondence and reprint requests to Dr. Dawson, 75 Francis Street, Boston, MA 02115.
Received April 21, 1987. Accepted for publication in final form May 31, 1988.
This article has been cited by other articles:
![]() |
S.-S. Lee and S.-H. Lee Does sensory Guillain-Barre syndrome exist without any abnormalities in motor nerve conduction? Neurology, March 28, 2006; 66(6): 947 - 948. [Full Text] [PDF] |
||||
![]() |
U Seneviratne and S Gunasekera Acute small fibre sensory neuropathy: another variant of Guillain-Barre syndrome? J. Neurol. Neurosurg. Psychiatry, April 1, 2002; 72(4): 540 - 542. [Abstract] [Full Text] [PDF] |
||||
![]() |
S. J. Oh, C. LaGanke, and G. C. Claussen Sensory Guillain-Barre syndrome Neurology, January 9, 2001; 56(1): 82 - 86. [Abstract] [Full Text] [PDF] |
||||
![]() |
U. Seneviratne Guillain-Barre syndrome Postgrad. Med. J., December 1, 2000; 76(902): 774 - 782. [Abstract] [Full Text] |
||||
| HOME | HELP | FEEDBACK | SUBSCRIPTIONS | ARCHIVE | SEARCH | TABLE OF CONTENTS |