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Department of Neurology, Division of Neurologic Rehabilitation, University of California, Los Angeles, CA.
Thirteen patients presented with brief, repetitive, stereotyped transient isehemic attacks, large artery atherostenoses or occlusions with impaired collateral flow to a cortical perfusion borderzone, and orthostatic hypotension (OH). OH was caused by diabetes mellitus, aging, and treatments for isehemic heart disease and hypertension. Medical management of OH often eliminated the need for stroke prevention measures such as surgery or anticoagulation. Focal cerebral hypoperfusion from the combination of occlusive vascular disease and OH may be an underreported, treatable cause of TIA and stroke.
Address correspondence and reprint requests to Dr. Dobkin, 301 North Prairie Avenue, Inglewood, CA 90301.
Presented in part at the fortieth annual meeting of the American Academy of Neurology, Cincinnati, OH, April 1988.
Received June 9, 1988. Accepted for publication in final form July 18, 1988.
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