Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Kincaid, J. C.
Right arrow Articles by Benson, M. D.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Kincaid, J. C.
Right arrow Articles by Benson, M. D.
NEUROLOGY 1989;39:861
© 1989 American Academy of Neurology

Late-onset familial amyloid polyneuropathy in an American family of English origin

John C. Kincaid, MD, Margaret R. Wallace, PhD and Merrill D. Benson, MD

Departments of Medicine, Medical Genetics and Neurology, Indiana University School of Medicine, and Richard L. Roudebush Veterans Administration Medical Center, Indianapolis, IN.

Recent advances permit biochemical and genetic classification of familial amyloidotic polyneuropathy. We report a family with the methio-nine-30 prealbumin variant, which is atypical in late age of onset and the appearance of proximal arm weakness before more typical generalized neuropathy.

Address correspondence and reprint requests to Dr. Benson, Richard L. Roudebush VA Medical Center, 1481 West 10th Street, Indianapolis, IN 46202.

Supported by VA Medical Research (MRIS 583-0888), and grants from RR-00750 (GCRC), United States Public Health Service, National Institute of Arthritis and Musculoskeletal and Skin Diseases (AR 20582 and AR 7448), the Arthritis Foundation, the Grace M. Showalter Trust, and the Marion E. Jacobson Fund.

Received August 17, 1988. Accepted for publication in final form December 28, 1988.




This article has been cited by other articles:


Home page
 Arch NeurolHome page
M. L. Fiszman, M. Di Egidio, K. C. Ricart, M. G. Repetto, L. N. Borodinsky, S. F. Llesuy, R. D. Saizar, P. L. Trigo, S. Riedstra, P. P. Costa, et al.
Evidence of Oxidative Stress in Familial Amyloidotic Polyneuropathy Type 1
Arch Neurol, April 1, 2003; 60(4): 593 - 597.
[Abstract] [Full Text] [PDF]

Home page
 Arch NeurolHome page
H. Koike, K.-i. Misu, S.-i. Ikeda, Y. Ando, M. Nakazato, E. Ando, M. Yamamoto, N. Hattori, G. Sobue, and for the Study Group for Hereditary Neuropathy in J
Type I (Transthyretin Met30) Familial Amyloid Polyneuropathy in Japan: Early- vs Late-Onset Form
Arch Neurol, November 1, 2002; 59(11): 1771 - 1776.
[Abstract] [Full Text] [PDF]

Home page
 Arch NeurolHome page
A. Yoshioka, Y. Yamaya, S. Saiki, G. Hirose, K. Shimazaki, M. Nakamura, and Y. Ando
A Case of Familial Amyloid Polyneuropathy Homozygous for the Transthyretin Val30Met Gene With Motor-Dominant Sensorimotor Polyneuropathy and Unusual Sural Nerve Pathological Findings
Arch Neurol, November 1, 2001; 58(11): 1914 - 1918.
[Abstract] [Full Text] [PDF]

Home page
 BrainHome page
K.-i. Misu, N. Hattori, M. Nagamatsu, S.-i. Ikeda, Y. Ando, M. Nakazato, Y.-i. Takei, N. Hanyu, Y. Usui, F. Tanaka, et al.
Late-onset familial amyloid polyneuropathy type I (transthyretin Met30-associated familial amyloid polyneuropathy) unrelated to endemic focus in Japan: Clinicopathological and genetic features
Brain, October 1, 1999; 122(10): 1951 - 1962.
[Abstract] [Full Text] [PDF]

Home page
 NEJMHome page
M. D. Benson
Aging, Amyloid, and Cardiomyopathy
N. Engl. J. Med., February 13, 1997; 336(7): 502 - 504.
[Full Text]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1989 by AAN Enterprises, Inc.