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NEUROLOGY 1989;39:1253
© 1989 American Academy of Neurology

Regional brain glucose metabolism in neuroacanthocytosis

Richard M. Dubinsky, MD, Mark Hallett, MD, Robert Levey, MD and Giovanni Di Chiro, MD

Human Motor Control Section, Medical Neurology Branch, and the Neuroimaging Section, Division of Intramural Research, National Institute of Neurological Disorders and Stroke, Bethesda, MD.

Two brothers with neuroacanthocytosis had [18F]-2-fluoro-2-deoxyglucose PET scans showing marked glucose hypometabolism of the caudate and putamen. MRIs showed no evidence of atrophy or modification of signal intensity in these structures. Decreased glucose utilization of the striatum can underlie hyperkinetic movement disorders of various etiologies.

Address correspondence and reprint requests to Dr. Hallett, Building 10, Room 5N226, NINDS, National Institutes of Health, Bethesda, MD 20892.

Received October 18, 1988. Accepted for publication in final form March 15, 1989.




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