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Antimyoclonic effects of alcohol in progressive myoclonus epilepsy

From Centre Saint Paul, Marseille, France.

We studied the effect of a single oral intake of 30 to 40 grams of alcohol in 4 consecutive patients presenting with Ramsay Hunt syndrome, the most common type of degenerative progressive myoclonic epilepsy (PME) encountered in southern Europe. Clinical and polygraphic monitoring demonstrated abatement of myoclonus in all patients, although the degree of improvement varied among patients. These findings show that alcohol is a potent antimyoclonic agent. In the particular context of PME, occasional alcohol intake may help patients in their social life.

Address correspondence and reprint requests to Dr. Pierre Genton, Centre Saint Paul, 300 Boulevard de Sainte Marguerite, 13009 Marseille, France.

Presented in part at the Workshop on Progressive Myoclonus Epilepsies, Marseilles, France, June 24, 198bt.

Received October 23, 1989. Accepted for publication in final form February 14, 1990.

This article has been cited by other articles:

				P. Genton, P. Gelisse, G. L. Krauss, A. Bergin, Y.-W. Cho, S. G. Reich, and R. E. Kramer Suppression of post-hypoxic and post-encephalitic myoclonus with levetiracetam Neurology, September 25, 2001; 57(6): 1144 - 1145. [Full Text] [PDF]