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Parietal sensory loss simulating a peripheral lesion, documented by somatosensory evoked potentials

Institute of Neurological Sciences, The Prince Henry and Prince of Wales Hospitals; and the School of Medicine, University of New South Wales, Sydney, Australia.

We describe "pseudoradicular" sensory impairment caused by parietal infarction and documented by abnormalities in the CT and somatosensory cortical responses evoked from median and ulnar territories.

Address correspondence and reprint requests to Dr. James W. Lance, Professor of Neurology, The Prince Henry Hospital, Little Bay, NSW 2036, Australia.

Received January 30, 1990. Accepted for publication in final form June 22, 1990.

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