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NEUROLOGY 1996;46:559-561
© 1996 American Academy of Neurology

Immune brachial plexus neuropathy

Suggestive evidence for an inflammatory-immune pathogenesis

G. A. Suarez, MD, C. Giannini, MD, E. P. Bosch, MD, R. J. Barohn, MD, J. Wodak, MBBS, FRACP, P. Ebeling, MBBS, FRACP, R. Anderson, MBBS, FRACP, P. E. McKeever, MD, PhD, M. B. Bromberg, MD, PhD and P. J. Dyck, MD

From the Peripheral Neuropathy Research Center, Department of Neurology (Drs. Suarez, Giannini, and Dyck), Mayo Clinic and Mayo Foundation, Rochester, MN; Mayo Clinic Scottsdale (Dr. Bosch), Scottsdale, AZ; University of Texas Southwestern Medical Center (Dr. Barohn), Dallas, TX; The Alfred Group of Hospitals (Drs. Wodak and Anderson), Prahian Victoria, Australia; Royal Melbourne Hospital (Dr. Ebeling), Melbourne, Australia, University of Michigan School of Medicine (Dr. McKeever), Ann Arbor, MI; and University of Utah (Dr. Bromberg), Salt Lake City, UT.
Supported in part by grants from the National Institute of Neurological and Communicative Disorders and Stroke (14304) and from the Muscular Dystrophy Association.
Received July 5, 1995. Accepted in final form July 27, 1995.
Address correspondence and reprint requests to Dr. Guillermo A. Suarez, Peripheral Nerve Center, Department of Neurology, Mayo Clinic, 200 First Street SW, Rochester, MN 55905.

We report brachial plexus biopsy findings from two Australian and two American patients with brachial plexus neuropathy.There were florid multifocal mononuclear inflammatory cell infiltrates. Present evidence suggests that these brachial neuropathies have an immune basis.

NEUROLOGY 1996;46: 559-561




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