Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Figures Only
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Amato, A. A.
Right arrow Articles by Shebert, R. T.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Amato, A. A.
Right arrow Articles by Shebert, R. T.
NEUROLOGY 1998;51:598-600
© 1998 American Academy of Neurology

Inclusion body myositis in twins

Anthony A. Amato, MD and Robert T. Shebert, MD

From the Department of Medicine/Neurology (Dr. Amato), University of Texas Health Science Center at San Antonio, and the Department of Neurology (Dr. Shebert), University of Miami School of Medicine, FL.

Address correspondence and reprint requests to Dr. Anthony A. Amato, Department of Medicine/Neurology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78284-7883.

Sporadic inclusion body myositis (s-IBM) is characterized by late onset of slowly progressive weakness that involves the quadriceps and volar forearm muscles early in the course of the disease. There are hereditary forms of inclusion body myopathy (h-IBM) that histologically resemble s-IBM. The lack of inflammation on biopsy and the different ages at onset and patterns of muscle weakness distinguish s-IBM from h-IBM. We report twin brothers with the typical clinical and histologic features of s-IBM. The occurrence of s-IBM in these twins suggests the possibility of a genetic susceptibility to developing s-IBM.

Received May 28, 1997. Accepted in final form March 27, 1998.




This article has been cited by other articles:


Home page
 BMJHome page
S. A Greenberg
How citation distortions create unfounded authority: analysis of a citation network
BMJ, July 23, 2009; 339(jul20_3): b2680 - b2680.
[Abstract] [Full Text] [PDF]

Home page
 Ann Rheum DisHome page
B Ranque-Francois, T Maisonobe, E Dion, J-C Piette, M-P Chauveheid, Z Amoura, and T Papo
Familial inflammatory inclusion body myositis
Ann Rheum Dis, April 1, 2005; 64(4): 634 - 637.
[Abstract] [Full Text] [PDF]

Home page
 NeurologyHome page
S. A. Greenberg, D. Sanoudou, J. N. Haslett, I. S. Kohane, L. M. Kunkel, A. H. Beggs, and A. A. Amato
Molecular profiles of inflammatory myopathies
Neurology, October 22, 2002; 59(8): 1170 - 1182.
[Abstract] [Full Text] [PDF]

Home page
 NeurologyHome page
M. Naumann, K. V. Toyka, A. A. Amato, and R. T. Shebert
Inclusion body myositis in twins
Neurology, August 1, 1999; 53(3): 656 - 656.
[Full Text]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1998 by AAN Enterprises, Inc.