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Neurology 1999;52:190
© 1999 American Academy of Neurology
Brief Communications

Dorsolateral infarction of the lower medulla: Clinical– MRI study

T. Brochier, PhD, M. Ceccaldi, MD, PhD, L. Milandre, MD and M. Brouchon, PhD

From the Centre de Recherche en Sciences Neurologiques (Dr. Brochier), Département de Physiologie, Université de Montréal, Canada; Département de Neuropsychologie (Drs. Brochier, Ceccaldi, and Brouchon), UFR de Médecine Université Aix-Marseille II; and Service de Neurologie (Drs. Ceccaldi and Milandre), Hôpital de la Timone Marseille, France.

Address correspondence and reprint requests to Dr. Thomas Brochier, Centre de Recherche en Sciences Neurologiques, Département de Physiologie, Université de Montréal, C.P. 6128 Succ. A, Montréal, Québec H3C 3J7, Canada.

We describe a man with lateral medullary syndrome associated with a long-standing clumsiness of the ipsilateral upper limb. MRI showed that the clinical finding of ipsilateral clumsiness correlated with an extension of the infarction into the dorsal column nuclei but was not reflected in any involvement of the more ventral pyramidal tract. This deficit in movement control that appears superficially like a hemiparesis may result from a combination of lemniscal and spinocerebellar deficits.




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J. S. Kim
Sensory symptoms in ipsilateral limbs/body due to lateral medullary infarction
Neurology, October 9, 2001; 57(7): 1230 - 1234.
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