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Neurology 2001;56:1597-1600
© 2001 American Academy of Neurology
Brief Communications

Hypothalamic amnesia with spontaneous confabulations: A clinicopathologic study

R. Ptak, PhD;, B. Birtoli, MSci;, H. Imboden, PhD;, C. Hauser, MD;, J. Weis, MD; and A. Schnider, MD

From the Department of Rehabilitation (Drs. Ptak and Schnider), University Hospital, Geneva; and Division for Neuropsychological Rehabilitation, University Hospital (Drs. Ptak and Schnider), and Division for Neurobiology (Drs. Birtoli and Imboden) and Neuropathology Unit, Institute of Pathology (Drs. Hauser and Weis), University of Bern, Switzerland.

Address correspondence and reprint requests to Prof. A. Schnider, Clinique de Rééducation, Hôpital Cantonal Universitaire, 26, av. de Beau-Séjour, CH-1211 Geneva 14, Switzerland; e-mail: armin.schnider{at}hcuge.ch

Previous studies demonstrated that patients producing spontaneous confabulations fail to suppress currently irrelevant memory traces and have anterior limbic lesions, particularly involving the orbitofrontal cortex or the basal forebrain. Here, a woman is described who had sarcoidosis damaging the medial hypothalamus and endocrine dysfunction, and a severe memory failure characterized by spontaneous confabulation, disorientation, and severely impaired free recall with preserved recognition. Isolated hypothalamic damage may produce the same type of memory disorder as orbitofrontal damage.




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