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Neurology 2001;57:534-537
© 2001 American Academy of Neurology
Brief Communications

Ketotic hyperglycemia and epilepsia partialis continua

F. Placidi, MD;, R. Floris, MD;, A. Bozzao, MD;, A. Romigi, MD;, M. E. Baviera, MD;, M. Tombini, MD;, F. Izzi, MD;, F. Sperli, MD; and M. G. Marciani, MD

From the Clinica Neurologica (Drs. Placidi, Romigi, Tombini, Izzi, Sperli, and Marciani) and Istituto di Radiologia (Drs. Floris and Bozzao), Università di Roma Tor Vergata; and Fondazione Santa Lucia (Drs. Placidi, Romigi, and Marciani), IRCCS, Rome, Italy.

Address correspondence and reprint requests to Dr. F. Placidi, Clinica Neurologica—Università di Roma Tor Vergata, P.le Umanesimo 10, 00144 Rome, Italy; e-mail: fbplacidi{at}libero.it

Epilepsia partialis continua (EPC) may occur during nonketotic hyperglycemia but has not been described with diabetic ketoacidosis. The authors report a patient with EPC associated with ketotic hyperglycemia. Brain MRI showed two areas of abnormal signal intensity in the left precentral gyrus and in the right cerebellar hemisphere. Hyperglycemia may reduce seizure threshold because of the increase in {gamma}-aminobutyric acid metabolism and may trigger epileptic discharges.




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