Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Figures Only
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow CME: Take the course for this article:
 Volume 57, Number 4, August 28, 2001
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Kusunoki, S.
Right arrow Articles by Kanazawa, I.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Kusunoki, S.
Right arrow Articles by Kanazawa, I.
Related Collections
Right arrow Bacterial infections
Right arrow Post-infectious
Right arrow Peripheral neuropathy
Right arrow Guillain-Barre syndrome
Neurology 2001;57:736-738
© 2001 American Academy of Neurology
Brief Communications

Anti-Gal-C antibodies in GBS subsequent to mycoplasma infection: Evidence of molecular mimicry

Susumu Kusunoki, MD;, Meiko Shiina, MD; and Ichiro Kanazawa, MD

From the Department of Neurology, Graduate School of Medicine, University of Tokyo, Japan.

Address correspondence and reprint requests to Dr. Susumu Kusunoki, Department of Neurology, Graduate School of Medicine, University of Tokyo, 7-3-1 Hongo, Bunkyo-ku, Tokyo 113-8655, Japan; e-mail: kusunoki-tky{at}umin.ac.jp

The authors previously reported the presence of antibody against galactocerebroside (Gal-C) in sera from patients with Guillain–Barré syndrome subsequent to Mycoplasma pneumoniae infection. Anti-Gal-C antibody activities in these sera were inhibited specifically by the M. pneumoniae reagent. A rabbit anti-Gal-C antibody recognized several glycolipids in M. pneumoniae. These data show that a Gal-C-like structure is present in M. pneumoniae, indicative of molecular mimicry between a major myelin glycolipid, Gal-C, and M. pneumoniae.




This article has been cited by other articles:


Home page
 J Trop PediatrHome page
B. Akyildiz, H. Gumus, S. Kumandas, A. Coskun, A. Baykan, A. Yikilmaz, I. Kara, and A. Okur
Guillain-Barre Syndrome Associated with Legionnella Infection
J Trop Pediatr, August 1, 2008; 54(4): 275 - 277.
[Abstract] [Full Text] [PDF]

Home page
 PediatricsHome page
L. J. Christie, S. Honarmand, D. F. Talkington, S. S. Gavali, C. Preas, C.-Y. Pan, S. Yagi, and C. A. Glaser
Pediatric Encephalitis: What Is the Role of Mycoplasma pneumoniae?
Pediatrics, August 1, 2007; 120(2): 305 - 313.
[Abstract] [Full Text] [PDF]

Home page
 NeurologyHome page
K. Susuki, M. Odaka, M. Mori, K. Hirata, and N. Yuki
Acute motor axonal neuropathy after Mycoplasma infection: Evidence of molecular mimicry
Neurology, March 23, 2004; 62(6): 949 - 956.
[Abstract] [Full Text] [PDF]

Home page
 BrainHome page
H. J. Willison and N. Yuki
Peripheral neuropathies and anti-glycolipid antibodies
Brain, December 1, 2002; 125(12): 2591 - 2625.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 2001 by AAN Enterprises, Inc.