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From the Department of Neurology (Dr. King), Royal Melbourne Hospital; and the Department of Radiology (Drs. Mitchell, Thomson, and Tress), University of Melbourne, Royal Melbourne Hospital, Victoria, Australia.
Address correspondence and reprint requests to Dr. John King, Box 2017, The Royal Melbourne Hospital, Victoria, Australia 3050; e-mail: jking{at}bigpond.net.au
Objective: To determine by cerebral venography and manometry in patients with idiopathic intracranial hypertension the cause of the previously demonstrated venous hypertension in the superior sagittal and proximal transverse sinuses.
Methods: Cerebral venous sinus pressure was measured before and immediately after C1-2 puncture with removal of 20 to 25 mL of CSF.
Results: Lowering the intracranial pressure by lateral C1-2 puncture during manometry has shown that the venous hypertension resolves immediately.
Conclusion: These studies indicate that the venous hypertension is due to compression of the transverse sinuses by raised intracranial pressure and not due to a primary obstructive process in the cerebral venous sinuses.
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