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Neurology 2002;58:653-655
© 2002 American Academy of Neurology
Brief Communications

Higher lipoprotein (a) levels in atherothrombotic than lacunar ischemic cerebrovascular disease

P. Cerrato, MD;, D. Imperiale, MD;, P. Fornengo, MD;, G. Bruno, MD;, M. Cassader, PhD;, P. Maffeis, MD;, P. Cavallo Perin, MD;, G. Pagano, MD; and B. Bergamasco, MD

From the Departments of Neuroscience (Drs. Cerrato, Imperiale, Maffeis, and Bergamasco) and Internal Medicine (Drs. Fornengo, Bruno, Cassader, Cavallo Perin, and Pagano), University of Turin, Torino, Italy.

Address correspondence and reprint requests to Dr. Paolo Cerrato, Department of Neuroscience, University of Torino; Via Cherasco 15, 10126 Torino, Italy; e-mail: paolo_cerrato{at}yahoo.com

To investigate the role of plasma lipid abnormalities in ischemic cerebrovascular disease related to primary vessel disease, the authors assess lipid profiles in a hospital-based cohort of 202 consecutive patients with atherothrombotic or lacunar stroke subtypes. Lipoprotein (a) was the unique lipid parameter that differs between these two subtypes being its value twofold higher in patients with atherothrombotic than in lacunar stroke. This suggests that lipoprotein (a) promotes large vessel atheromatosis rather than small vessel arteriolosclerosis and favors thrombosis on atheromatous plaques by suppressing local fibrinolysis.




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