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Neurology 2003;60:1690-1692
© 2003 American Academy of Neurology
Brief Communications

Cardiac troponin levels following monitored epileptic seizures

B. K. Woodruff, MD, J. W. Britton, MD, S. Tigaran, MD PhD, G. D. Cascino, MD, M. F. Burritt, PhD, J. P. McConnell, PhD, J. Ravkilde, DMSc MD, H. Molgaard, DMSc MD, F. Andreasen, DMSc MD, M. Dam, DMSc MD and A. S. Jaffe, MD

From the Departments of Neurology (Drs. Woodruff, Britton, and Cascino), Laboratory Medicine and Pathology (Drs. Burritt, McConnell, and Jaffe), and Cardiology (Dr. Jaffe), Mayo Clinic Rochester, MN; the Departments of Neurology (Drs. Tigaran and Dam), Cardiology (Dr. Molgaard), and Pharmacology (Dr. Andreasen), Aarhus University Hospital; and the Department of Cardiology (Dr. Ravkilde), Aalborg University Hospital, Denmark.

Address correspondence and reprint requests to Dr. Jeffrey W. Britton, Department of Neurology, W8B Mayo Building, Mayo Clinic, Rochester, MN 55905; e-mail: britton.jeffrey{at}mayo.edu

Cardiac arrhythmia associated with myocardial injury is a proposed mechanism for sudden unexplained death in epilepsy. The authors measured serial cardiac troponin levels in 11 patients after monitored seizures. Using highly sensitive assays and criteria, no troponin elevations were seen, indicating that myocardial injury does not occur during uncomplicated seizures. An elevation in postictal troponin elevations should suggest the presence of cardiac injury secondary to neurocardiogenic mechanisms or primary cardiac factors, prompting further evaluation.




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C. Stollberger and J. Finsterer
Cardiac troponin levels following monitored epileptic seizures
Neurology, April 27, 2004; 62(8): 1453 - 1453.
[Full Text] [PDF]

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Cardiac troponin levels following monitored epileptic seizures
Claudia Stollberger, et al.
Neurology Online, 19 Aug 2003 [Full text]

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