HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Figures Only Full Text Full Text (PDF) Correspondence: Submit a response Alert me when this article is cited Alert me when Correspondence are posted Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Alert me to new issues of the journal Download to citation manager Citing Articles Citing Articles via HighWire Citing Articles via Google Scholar Google Scholar Articles by Hilton, D. A. Articles by Footitt, D. Search for Related Content PubMed Articles by Hilton, D. A. Articles by Footitt, D. Related Collections All Immunology All Cerebrovascular disease/Stroke Infarction

Neuropathological findings in Sneddon’s syndrome

From the Department of Histopathology (Dr. Hilton), Derriford Hospital, Plymouth; and the Department of Neurology (Dr. Footitt), Royal Devon and Exeter Hospital, Exeter, UK.

Address correspondence and reprint requests to Dr. David Hilton, Department of Histopathology, Derriford Hospital, Plymouth PL6 8DH, UK; e-mail: david.hilton{at}phnt.swest.nhs.uk

The authors report the neuropathologic findings in a case of Sneddon’s syndrome. There were multiple small, predominantly cortical, infarcts, with focal hyperplasia and fibrotic occlusion of arterial vessels in the superficial white matter, cortex, and leptomeninges. A very occasional arterial thrombus was seen. These findings suggest that Sneddon’s syndrome is caused by a noninflammatory arteriopathy affecting superficial cerebral vessels.

This article has been cited by other articles:

				Y. Aladdin, M. Hamadeh, and K. Butcher The Sneddon Syndrome Arch Neurol, June 1, 2008; 65(6): 834 - 835. [Full Text] [PDF]