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NEUROLOGY 2004;62:635-636
© 2004 American Academy of Neurology
Brief Communications

Confirmation of the protective effect of iNOS in an independent cohort of Parkinson disease

S. Hague, PhD, T. Peuralinna, BS, J. Eerola, MD, O. Hellström, MD, P. J. Tienari, MD PhD and A. B. Singleton, PhD

From the Laboratory of Neurogenetics (Drs. Hague and Singleton), National Institute on Aging, NIH, Bethesda, MD; Department of Neurology (Drs. Peuralinna, Eerola, and Tienari), Helsinki University Central Hospital and University of Helsinki, Biomedicum-Helsinki, Neuroscience Programme, Helsinki; and Department of Neurology (Dr. Hellström), Seinäjoki Central Hospital, Seinäjoki, Finland.

Address correspondence and reprint requests to Dr. Singleton, Laboratory of Neurogenetics, National Institute on Aging, NIH, Building 10 Room 6C103, 9000 Rockville Pike, Bethesda, MD 20892; e-mail: singleta{at}mail.nih.gov

Nitric oxide is a biologic messenger molecule involved in a diverse range of physiologic processes. An exon 22 inducible nitric oxide synthase genotype has recently been reported to be protective against Parkinson disease in a European cohort. The authors confirm the protective effect of this genotype (OR = 0.5, 95% CI 0.27 to 0.93) in an independent Finnish case-control series.

Received July 30, 2003. Accepted in final form October 6, 2003.




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C. Schulte, M. Sharma, J. C. Mueller, P. Lichtner, J. Prestel, D. Berg, and T. Gasser
Comprehensive association analysis of the NOS2A gene with Parkinson disease
Neurology, December 12, 2006; 67(11): 2080 - 2082.
[Abstract] [Full Text] [PDF]


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