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NEUROLOGY 2004;62:984-986
© 2004 American Academy of Neurology
Brief Communications

Rasmussen encephalitis with active inflammation and delayed seizures onset

I. Korn-Lubetzki, MD, C. G. Bien, MD, J. Bauer, PhD, M. Gomori, MD, H. Wiendl, MD, L. Trajo, MD, H. Ovadia, PhD, B. Wilken, MD, V. H. Hans, MD, C. E. Elger, MD FRCP, H. Hurvitz, MD and I. Steiner, MD

From the Neurological Service (Dr. Korn-Lubetzki) and the Department of Pediatrics (Dr. Hurvitz), Bikur Cholim Hospital, Jerusalem, Israel; Departments of Epileptology (Drs. Bien and Elger) and Neuropathology (Dr. Hans), University of Bonn, Germany; Division of Neuroimmunology (Dr. Bauer), Brain Research Institute, Vienna, Austria; Departments of Neuroradiology (Dr. Gomori) and Neurology (Drs. Steiner and Ovadia), Hadassah University Hospital, Jerusalem, Israel; Department of Neurology (Dr. Wiendl), University of Tübingen, Germany; Department of Pathology (Dr. Trajo), Tel-Aviv Sourasky Medical Center, Israel; and Department of Pediatric Neurology (Dr. Wilken), Klinikum Kassel, Germany.

Address correspondence and reprint requests to Dr. I. Korn-Lubetzki, Neurological Service, Bikur Cholim Hospital, Strauss Street 5, P.O. Box 492, Jerusalem 91004, Israel; e-mail: ikl{at}md.huji.ac.il

Severe focal epilepsy is regarded as a clinical hallmark of Rasmussen encephalitis (RE). The authors report two children with progressive hemiparesis, contralateral hemispheric atrophy, and pathologic features characteristic for RE. At histologic diagnosis and over several months, neither patient experienced seizures. The report enlarges the clinical spectrum of RE and suggests that seizures are not an obligatory presenting symptom of the disorder.

Received August 1, 2003. Accepted in final form November 13, 2003.

Drs. Korn-Lubetzki and Bien contributed equally to the manuscript.




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