Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Koponen, S.
Right arrow Articles by Tenovuo, O.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Koponen, S.
Right arrow Articles by Tenovuo, O.
Related Collections
Right arrow All Neuropsychology/Behavior
Right arrow All Cognitive Disorders/Dementia
Right arrow Brain trauma
NEUROLOGY 2004;63:749-750
© 2004 American Academy of Neurology


Brief Communications

APOE-{epsilon}4 predicts dementia but not other psychiatric disorders after traumatic brain injury

S. Koponen, MD, T. Taiminen, MD PhD, V. Kairisto, MD PhD, R. Portin, PhD, H. Isoniemi, MD, S. Hinkka, PhLic and O. Tenovuo, MD PhD

From the Departments of Psychiatry (Drs. Koponen and Taiminen), Clinical Chemistry and Hematology (Dr. Kairisto), and Neurology (Drs. Portin, Isoniemi, and Tenovuo), Turku University Hospital, Finland; and the Department of Biostatistics (Dr. Hinkka), University of Turku, Finland.

Address correspondence and reprint requests to Dr. Salla Koponen, Department of Psychiatry, Turku University Hospital, PL 52, FIN-20521 Turku, Finland; e-mail: salla.koponen{at}utu.fi

The authors studied the association between APOE-{epsilon}4 genotype and axis I and II psychiatric disorders an average of 30 years after traumatic brain injury. Sixty patients were dichotomized into subjects with and without APOE-{epsilon}4 allele. Dementia and subclinical dementia were significantly more common with the presence of APOE-{epsilon}4. The occurrence of other psychiatric disorders did not differ between patients with and without APOE-{epsilon}4 allele.


Received October 1, 2003. Accepted in final form April 20, 2004.




This article has been cited by other articles:


Home page
NeurologyHome page
H. Isoniemi, T. Kurki, O. Tenovuo, V. Kairisto, and R. Portin
Hippocampal volume, brain atrophy, and APOE genotype after traumatic brain injury.
Neurology, September 12, 2006; 67(5): 756 - 760.
[Abstract] [Full Text] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 2004 by AAN Enterprises, Inc.