Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Figures Only
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Bender, A.
Right arrow Articles by Straube, A.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Bender, A.
Right arrow Articles by Straube, A.
Related Collections
Right arrow Meningitis
Right arrow All Toxicology
NEUROLOGY 2004;63:1311-1313
© 2004 American Academy of Neurology
Brief Communications

Severe symptomatic aseptic chemical meningitis following myelography

The role of procalcitonin

Andreas Bender, MD, Matthias Elstner, MD, Robert Paul, MD and Andreas Straube, MD

From the Department of Neurology, University of Munich, Klinikum Grosshadern, Germany.

Address correspondence and reprint requests to Dr. Andreas Bender, Department of Neurology, Klinikum Grosshadern, Marchioninistr. 15, 81377 Munich, Germany; e-mail: andreas.bender{at}med.uni-muenchen.de

Symptomatic aseptic, chemical meningitis is a rare complication of myelography. Its acute clinical course and standard laboratory findings are indistinguishable from those of bacterial meningitis. The authors present a case of severe postmyelographic chemical meningitis and compare CSF and serum inflammatory markers to a group of seven patients with proven bacterial meningitis. As in viral meningitis, procalcitonin might be able to discriminate between bacterial and chemical causes of CNS inflammation.

Received April 13, 2004. Accepted in final form May 26, 2004.




This article has been cited by other articles:


Home page
 Canadian J. AnesthesiaHome page
J. Mizuno, T. Gauss, M. Suzuki, M. Hayashida, H. Arita, and K. Hanaoka
Encephalopathy and rhabdomyolysis induced by iotrolan during epiduroscopy: [Encephalopathie et rhabdomyolyse provoquees par l'iotrolan durant l'epiduroscopie]
Can J Anesth, January 1, 2007; 54(1): 49 - 53.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 2004 by AAN Enterprises, Inc.