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NEUROLOGY 2005;64:2126-2128
© 2005 American Academy of Neurology
Brief Communications

Drop attacks secondary to superior canal dehiscence syndrome

Krister Brantberg, MD, Akira Ishiyama, MD and Robert W. Baloh, MD

From the Department of Audiology (Dr. Brantberg), Karolinska University Hospital, Stockholm, Sweden; and the Departments of Neurology (Dr. Baloh) and Surgery (Head & Neck) (Drs. Ishiyama and Baloh), UCLA.

Address correspondence and reprint requests to Dr. Robert W. Baloh, Department of Neurology, UCLA, Box 951769, Los Angeles, CA 90095-1769; e-mail: rwbaloh{at}ucla.edu

Two patients with unprovoked drop attacks were found to have dehiscence of the superior semicircular canal on CT of the temporal bone. Both had conductive hearing loss, preservation of stapedius reflex, and abnormal vestibular evoked myogenic potentials. Neither had sound- or pressure-induced nystagmus. Repair of the dehiscence in one case stopped the drop attacks, supporting a causal relationship between the dehiscence and the drop attacks.

Drs. Baloh and Ishiyama are supported by NIH grants AG 09693 and P50 DC 05224.

Received January 25, 2005. Accepted in final form March 3, 2005.




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[Abstract] [Full Text] [PDF]


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