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 Volume 64, Number 4, February 22, 2005
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NEUROLOGY 2005;64:716-718
© 2005 American Academy of Neurology
Brief Communications

A topodiagnostic investigation on body lateropulsion in medullary infarcts

F. Thömke, MD, J. J. Marx, MD, G. D. Iannetti, MD, PhD, G. Cruccu, MD, S. Fitzek, MD, P. P. Urban, MD, P. Stoeter, MD, M. Dieterich, MD and H. C. Hopf, MD

From the Department of Neurology (Drs. Thömke, Marx, Urban, Dieterich, and Hopf) and the Institute of Neuroradiology (Dr. Stoeter), University of Mainz, Germany; Department of Neurological Sciences (Drs. Iannetti and Cruccu), Università degli Studi di Roma "La Sapienza," Rome, Italy; and Department of Neurology (Dr. Fitzek), University of Jena, Germany.

Address correspondence and reprint requests to Dr. Frank Thömke, Department of Neurology, University of Mainz, Langenbeckstr. 1, D-55101 Mainz, Germany; e-mail: thoemke{at}neurologie.klinik.uni-mainz.de

Body lateropulsion may occur without signs of vestibular dysfunction and vestibular nucleus involvement. The authors examined 10 such patients with three-dimensional brainstem mapping. Body lateropulsion without limb ataxia reflected an impairment of vestibulospinal postural control caused by a lesion of the descending lateral vestibulospinal tract, whereas body lateropulsion with limb ataxia was probably the consequence of impaired or absent proprioceptive information caused by a lesion of the ascending dorsal spino-cerebellar tract.




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