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NEUROLOGY 2005;65:1971-1973
© 2005 American Academy of Neurology
Brief Communications

Dopamine transporter imaging study in parkinsonism occurring in fragile X premutation carriers

R. Ceravolo, MD, A. Antonini, MD, D. Volterrani, MD, C. Rossi, MD, S. Goldwurm, MD, E. Di Maria, MD, L. Kiferle, MD, U. Bonuccelli, MD and L. Murri, MD

From the Department of Neurosciences, Section of Neurology (Drs. Ceravolo, Rossi, Kiferle, Bonuccelli, and Murri), University of Pisa, Italy; Parkinson Institute (Drs. Antonini and Goldwurm), Istituti Clinici di Perfezionamento, Milan, Italy; Center of Nuclear Medicine (Dr. Volterrani), University of Pisa, Italy; Department of Neurosciences, Ophthalmology & Genetics, Section of Medical Genetics (Dr. Di Maria), University of Genova, Italy.

Address correspondence and reprint requests to Dr. R. Ceravolo, Department of Neurosciences, Section of Neurology, University of Pisa, via Roma 67, 56126 Pisa, Italy; e-mail: r.ceravolo{at}med.unipi.it

The authors studied four patients with parkinsonism carrying the fragile X premutation using SPECT with [23I]FP-CIT. They found evidence of preserved presynaptic nigrostriatal function, suggesting that parkinsonism in the X fragile premutation might be related to postsynaptic dopaminergic changes or different neurotransmitter alterations.

Disclosure: The authors report no conflicts of interest.

Received March 3, 2005. Accepted in final form September 13, 2005.


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