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NEUROLOGY 2007;68:2129-2131
© 2007 American Academy of Neurology

Statin enhancement of collateralization in acute stroke

B. Ovbiagele, MD, J. L. Saver, MD, S. Starkman, MD, D. Kim, MD, L. K. Ali, MD, R. Jahan, MD, G. R. Duckwiler, MD, F. Viñuela, MD, S. Pineda, BS and D. S. Liebeskind, MD

From the Departments of Neurology (B.O., J.L.S., S.S., D.K., L.K.A., S.P., D.S.L.), Emergency Medicine (S.S.), and Radiology (R.J., G.R.D., F.V.), UCLA Medical Center, Los Angeles, CA.

Address correspondence and reprint requests to Dr. Bruce Ovbiagele, UCLA Stroke Center and Department of Neurology, 710 Westwood Plaza, Los Angeles, CA 90095 ovibes{at}mednet.ucla.edu

Collateral circulation influences cerebral infarction occurrence and size. Statins may improve ischemic stroke outcomes. We evaluated the relationship between prestroke statin use and pretreatment angiographic collateral grade among acute ischemic stroke patients presenting with occlusion of a major cerebral artery. After adjusting for covariates, the statin-treated group had significantly higher collateral scores than non–statin users, suggesting an association between statin use and better collateralization during acute stroke.

Supported by NIH/NINDS 1K23NS054084-01A1 (D.S.L.).

Disclosure: The authors report no conflicts of interest.

Received November 7, 2006. Accepted in final form February 1, 2007.






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