Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


This Article
Right arrow Figures Only
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Barroso, B.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Barroso, B.
Related Collections
Right arrow Stroke in young adults
Right arrow Arteriovenous malformation
NEUROLOGY 2008;71:e42
© 2008 American Academy of Neurology


Resident and Fellow Section

Teaching NeuroImage: Pulmonary arteriovenous fistula and top of the basilar infarction

Bruno Barroso, MD

From the Service de Neurologie, Centre Hospitalier François Mitterrand, Pau cedex, France.

Address correspondence and reprint requests to Dr. Bruno Barroso, Service de Neurologie, Centre Hospitalier François Mitterrand, 4 Boulevard Hauterive, 64046 Pau cedex, France

A 25-year-old woman presented with acute unconsciousness. Examination revealed bilateral Babinski signs, fluctuating pupillary abnormalities (alternately widely dilated to miotic), upgaze paralysis, and multidirectional nystagmus.

Brain MRI sequences at 3 hours after onset revealed bilateral hyperintense signals in the thalami, consistent with infarcts (figure, A), as well as in the distal basilar trunk and posterior cerebral arteries (figure, B), consistent with intravascular thrombi. MR angiography confirmed occlusion of both posterior cerebral arteries.


Figure 118
View larger version (34K):
[in this window]
[in a new window]

 
Figure Brain MRI 3 hours after onset (A, B) and chest CT (C)

(A) Axial diffusion-weighted MRI shows high-intensity signals in the bilateral medial thalami. (B) Axial FLAIR MRI shows high- intensity signals in the distal basilar and posterior cerebral arteries. (C) Chest CT demonstrates two left pulmonary arteriovenous fistulas.

 

No hypercoagulable state, cardioembolic source, or dissection was identified. There was a family history of hemorrhagic telangiectasia (Osler-Weber-Rendu syndrome), and chest CT scan demonstrated pulmonary arteriovenous fistulas (figure, C). Deep venous thrombosis was not identified.

Paradoxical embolization is considered the likely predominant mechanism of cerebral ischemia in patients with pulmonary arteriovenous fistulas.1


Disclosure: The author reports no disclosures.

bruno.barroso{at}ch-pau.fr


    REFERENCE
 Top.
 REFERENCE
 

  1. Moussouttas M, Fayad P, Rosenblatt M, et al. Pulmonary arteriovenous malformations: cerebral ischemia and neurologic manifestations. Neurology 2000;55:959–964.[Abstract/Free Full Text]




This Article
Right arrow Figures Only
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Barroso, B.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Barroso, B.
Related Collections
Right arrow Stroke in young adults
Right arrow Arteriovenous malformation


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS