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 Volume 67, Number 9, November 14, 2006
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NEUROLOGY 2006;67:1668-1670
© 2006 American Academy of Neurology
Brief Communications

Clinical and radiographic features of peritumoral infarction following resection of glioblastoma

S. Ulmer, MD*, T. A. Braga, MD*, F. G. Barker, II, MD, M. H. Lev, MD, R. G. Gonzalez, MD, PhD and J. W. Henson, MD

From Stephen E. and Catherine Pappas Center for Neuro-oncology (S.U., T.A.B., F.G.B., J.W.H.), Division of Neuroradiology (S.U., T.A.B., M.H.L., R.G.G., J.W.H.), Neurosurgical Service (F.G.B.), Massachusetts General Hospital; and Harvard Medical School (F.G.B., M.H.L., R.G.G., J.W.H.), Boston, MA.

Address correspondence and reprint requests to Dr. John W. Henson, Stephen E. Catherine Pappas Center for Neuro-oncology and Division of Neuroradiology, Massachusetts General Hospital, Yawkey 9 East, 55 Fruit Street, Boston, MA 02114; e-mail: henson{at}helix.mgh.harvard.edu

Focal areas of restricted diffusion adjacent to high-grade glioma resection cavities were detected in 70% of patients on immediate postoperative MRI studies. Follow-up studies demonstrated cystic encephalomalacia in 91% of these foci, suggesting the presence of infarction, and the infarcted tissue demonstrated enhancement in 43% of cases. New postoperative deficits correlated well with the anatomic region of infarction in six patients. Enhancement in perioperative infarcts can mimic tumor progression on follow-up imaging studies.

Editorial, see page 1540

*These authors contributed equally to this work.

S.U. and T.A.B. were supported by the Pappas Brain Tumor Imaging Program at the Massachusetts General Hospital.

Part of the data were presented in abstract form at the 2003 annual meeting of the American Academy of Neurology.

Disclosure: The authors report no conflicts of interest.

Received March 17, 2006. Accepted in final form July 10, 2006.


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