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February 13, 2006

Pure myopathy associated with a novel mitochondrial tRNA gene mutation

February 14, 2006 issue
66 (3) 447-449

Abstract

The authors describe a 47-year-old man who presented with proximal muscle weakness, myalgia, elevated creatine kinase, and features of a pure myopathic syndrome in whom they have identified a novel mutation in the mitochondrial tRNAAla gene. This 5591G>A transition is heteroplasmic, segregates with cytochrome c oxidase deficiency in single muscle fibers, and fulfills recognized criteria for pathogenicity. This case exemplifies the wide-ranging clinical spectrum of mitochondrial disease presentations.

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Information & Authors

Information

Published In

Neurology®
Volume 66Number 3February 14, 2006
Pages: 447-449

Publication History

Published online: February 13, 2006
Published in print: February 14, 2006

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Authors

Affiliations & Disclosures

H. Swalwell, BSc
From the Mitochondrial Research Group (H.S., D.M.T., R.W.T.), School of Neurology, Neurobiology and Psychiatry, The Medical School, University of Newcastle upon Tyne, UK; and Department of Neurology (M.D., H.H., M.S., S.Z.), Martin-Luther-Universität Halle-Wittenberg, Halle/Saale, Germany.
M. Deschauer, MD
From the Mitochondrial Research Group (H.S., D.M.T., R.W.T.), School of Neurology, Neurobiology and Psychiatry, The Medical School, University of Newcastle upon Tyne, UK; and Department of Neurology (M.D., H.H., M.S., S.Z.), Martin-Luther-Universität Halle-Wittenberg, Halle/Saale, Germany.
H. Hartl, MD
From the Mitochondrial Research Group (H.S., D.M.T., R.W.T.), School of Neurology, Neurobiology and Psychiatry, The Medical School, University of Newcastle upon Tyne, UK; and Department of Neurology (M.D., H.H., M.S., S.Z.), Martin-Luther-Universität Halle-Wittenberg, Halle/Saale, Germany.
M. Strauss, MD
From the Mitochondrial Research Group (H.S., D.M.T., R.W.T.), School of Neurology, Neurobiology and Psychiatry, The Medical School, University of Newcastle upon Tyne, UK; and Department of Neurology (M.D., H.H., M.S., S.Z.), Martin-Luther-Universität Halle-Wittenberg, Halle/Saale, Germany.
D. M. Turnbull, FRCP
From the Mitochondrial Research Group (H.S., D.M.T., R.W.T.), School of Neurology, Neurobiology and Psychiatry, The Medical School, University of Newcastle upon Tyne, UK; and Department of Neurology (M.D., H.H., M.S., S.Z.), Martin-Luther-Universität Halle-Wittenberg, Halle/Saale, Germany.
S. Zierz, MD
From the Mitochondrial Research Group (H.S., D.M.T., R.W.T.), School of Neurology, Neurobiology and Psychiatry, The Medical School, University of Newcastle upon Tyne, UK; and Department of Neurology (M.D., H.H., M.S., S.Z.), Martin-Luther-Universität Halle-Wittenberg, Halle/Saale, Germany.
R. W. Taylor, PhD
From the Mitochondrial Research Group (H.S., D.M.T., R.W.T.), School of Neurology, Neurobiology and Psychiatry, The Medical School, University of Newcastle upon Tyne, UK; and Department of Neurology (M.D., H.H., M.S., S.Z.), Martin-Luther-Universität Halle-Wittenberg, Halle/Saale, Germany.

Notes

Address correspondence and reprint requests to Dr. Robert W. Taylor, Mitochondrial Research Group, School of Neurology, Neurobiology and Psychiatry, The Medical School, University of Newcastle upon Tyne, Newcastle upon Tyne, NE2 4HH, UK; e-mail: [email protected]

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